P5151
Am J Respir Crit Care Med 2000 Sep;162(3 Pt 1):1052-7
Lung volume reduction surgery does not improve diaphragmatic contractile
properties or atrophy in hamsters with elastase-induced emphysema.
Marchand E, De Leyn P, Gayan-Ramirez G, Palecek F, de Bock V, Dom R,
Decramer M.
Respiratory Muscle Research Unit, Laboratory of Pneumology and
Respiratory Division, Thoracic Surgery Division, and Neuropathology
Department, University Hospitals, Katholieke Universiteit Leuven,
Leuven, Belgium.
It is claimed that lung volume reduction surgery (LVRS) improves
inspiratory muscle function. As diaphragm structure and function are not
directly appraisable in patients, we studied the effects of LVRS on the
diaphragm in vitro contractile properties and morphology in hamsters
with elastase-induced emphysema. Four months after intratracheal
instillation of elastase (40 U/100 g), hamsters underwent either
bilateral LVRS (LVRS, n = 11) or a sham operation (SHAM, n = 8). Four
animals died during the perioperative period in LVRS (n = 7). Hamsters
instilled with saline served as control (CTL, n = 8). Animals were
studied at the age of 9 mo. LVRS was associated with a significant 25%
decrease in functional residual capacity compared to SHAM (p < 0.05).
Compared with CTL, LVRS and SHAM showed a significant 18% and 14%
reduction in diaphragm mass, respectively (p = 0.02). LVRS had a
significantly decreased twitch tension compared to CTL and SHAM (p <
0.01). Both LVRS and SHAM showed increased resistance to muscle fatigue
compared with CTL. The histochemical analysis revealed a significant
shift from type IIx/b toward type IIa fibers in LVRS and SHAM compared
with CTL. In conclusion, emphysema is associated with functional
adaptations but LVRS does not appear to beneficially alter the diaphragm
contractile and morphological characteristics in hamsters with
elastase-induced emphysema.